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EternaGen / Research use onlyPeptide · Research use only
Semax
$54CAD
An ACTH-fragment-derived heptapeptide explored in intranasal neurobiology and neurotrophic-signalling research.
Available to approved purchasers
Select strength
1
Documentation review pending Fulfilled in Canada Research use only
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Contact supportProduct information
Semax, clearly explained.
An ACTH-fragment-derived heptapeptide explored in intranasal neurobiology and neurotrophic-signalling research.
Overview
Semax has been examined in small human neuroimaging and clinical studies, while much of the mechanistic literature comes from animal models. Those findings do not establish the safety, efficacy or performance of a separately sourced research material.
Semax is a seven-amino-acid synthetic peptide with the sequence Met-Glu-His-Phe-Pro-Gly-Pro. It is derived from an adrenocorticotropic-hormone fragment and has been studied through intranasal administration in a small number of human and animal experiments.- Format
- Nasal spray
- Available strengths
- 10mg
- Documentation
- Source review pending
Research at a glanceLimited small human studies plus preclinical mechanistic evidence
Reviewed 31 July 2026View research context Specifications
Product details
- Sequence / identity
- MEHFPGP
- Molecular formula
- C37H51N9O10S
- Molecular weight
- 813.9 g/mol
- Storage and handling
- Launch handling specifications will be published with the verified batch record. Protect the sealed research material from direct light in the interim.
Batch documentationSource review pending
Not yet publishedSupplier-origin documentation will be published only after EternaGen verifies the source and record details.
Important research-use boundary
This catalogue entry describes a research material, not an approved therapeutic formulation. Published findings do not provide directions for personal use and cannot verify the safety or performance of a separately sourced material.
Explore the research
What has been studied?
Keep the context attached
Check what was studied, what researchers observed, and the limitation attached to each finding.
Question 01
Resting-state brain networks
A small placebo comparison in healthy volunteers measured default-mode-network topology shortly after intranasal exposure.
Evidence so far: Small exploratory human studyQuestion 02
Neurotrophic signalling
Rat studies have examined specific binding and short-term changes in brain-derived neurotrophic factor after intranasal administration.
Evidence so far: Preclinical mechanistic evidenceQuestion 03
Translation and safety
The available literature does not establish long-term safety, broadly applicable benefit or equivalence between research materials and studied formulations.
Evidence so far: Major evidence gapProposed mechanism
How researchers are investigating the mechanism.
Animal research has examined Semax binding and changes in brain-derived neurotrophic factor in specific brain regions. A small human fMRI experiment measured short-term changes in resting-state network topology. These observations are exploratory and do not establish a clinical outcome.
Evidence gaps
What remains unknown?
- Larger independently replicated human trials with prespecified clinical outcomes are lacking.
- Long-term safety, formulation comparability and route-specific tolerability remain insufficiently characterized.
- A published experiment cannot authenticate the identity or performance of a separate catalogue material.
Primary sources
Check the original sources.
- Primary exploratory human studyPanikratova et al. Effects of Semax on the Default Mode Network of the Brain. Bull Exp Biol Med. 2018. PMID 30225715.
- Primary preclinical studyDolotov et al. Semax binding and BDNF protein in rat basal forebrain. J Neurochem. 2006. PMID 16635254.
- Compound identity recordPubChem CID 9811102. Semax molecular identity and sequence record.
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